In 1997, Haider and Moosa investigated the presence of survival motor neuron gene and neuronal apoptosis inhibitory protein gene deletions in 17 Arab and 1 Indian families with spinal muscular atrophy (15 type I and 3 type II). In a patient with spinal muscular atrophy type II, exons 7 and 8 of the survival motor neuron gene and exon 5 of the neuronal apoptosis inhibitory protein gene were deleted. This patient also had the Pierre Robin syndrome. No deletion was detected in healthy siblings or the parents.

Sawardekar (2005) conducted a study to establish the prevalence of major congenital malformations in children born during a 10-year period in an Omani hospital in Nizwa. Of the 21,988 total births in the hospital, four children were born with Pierre Robin Sequence.

Aziza et al. (2011) conducted a hospital-based descriptive study during 2002 to 2009 in the Cleft Lip/Palate and Craniofacial Anomalies Registry at King Faisal Specialist Hospital and Research Center (Riyadh). Of the 447 craniofacial patients (male, 242; female, 205), 109 (24%) had only cranial anomalies, 261 (58%) had only facial anomalies and 77 (17%) had both of these conditions. Among facial syndromes, 72 (59%) were observed to have Pierre-Robin sequence.

Hosani and Czeizel (2000) evaluated the pilot dataset [March-May 1998] of the UAE National Congenital Abnormality Registry (NCAR). A total of 4,861 births were recorded in this study period, with a birth prevalence of total congenital anomalies being 30.3/1,000 births. Pierre Robin syndrome was identified in three neonates, resulting in an incidence rate of 0.62/1,000 births.